forked from D-Net/dnet-hadoop
5407 lines
251 KiB
XML
5407 lines
251 KiB
XML
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<Title>Biochemical and biophysical research communications</Title>
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<ArticleTitle>Delineation of the intimate details of the backbone conformation of pyridine nucleotide
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coenzymes in aqueous solution.
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<LastName>Barthel</LastName>
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<Chemical>
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<RegistryNumber>436O5HM03C</RegistryNumber>
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<NameOfSubstance UI="D004087">Dihydroergotamine</NameOfSubstance>
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</Chemical>
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<Chemical>
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<DateCompleted>
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<Abstract>
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<AbstractText>(--)-alpha-Bisabolol has a primary antipeptic action depending on dosage, which is not
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only occurs in case of direct contact. In case of a previous contact with the substrate, the
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|
</Abstract>
|
|
<AuthorList CompleteYN="Y">
|
|
<Author ValidYN="Y">
|
|
<LastName>Isaac</LastName>
|
|
<ForeName>O</ForeName>
|
|
<Initials>O</Initials>
|
|
</Author>
|
|
<Author ValidYN="Y">
|
|
<LastName>Thiemer</LastName>
|
|
<ForeName>K</ForeName>
|
|
<Initials>K</Initials>
|
|
</Author>
|
|
</AuthorList>
|
|
<Language>ger</Language>
|
|
<PublicationTypeList>
|
|
<PublicationType UI="D004740">English Abstract</PublicationType>
|
|
<PublicationType UI="D016428">Journal Article</PublicationType>
|
|
</PublicationTypeList>
|
|
<VernacularTitle>Biochemische Untersuchungen von Kamilleninhaltsstoffen. III. In-vitro-Versuche über die
|
|
antipeptische Wirkung des (-)-alpha-Bisabolols
|
|
</VernacularTitle>
|
|
</Article>
|
|
<MedlineJournalInfo>
|
|
<Country>Germany</Country>
|
|
<MedlineTA>Arzneimittelforschung</MedlineTA>
|
|
<NlmUniqueID>0372660</NlmUniqueID>
|
|
<ISSNLinking>0004-4172</ISSNLinking>
|
|
</MedlineJournalInfo>
|
|
<ChemicalList>
|
|
<Chemical>
|
|
<RegistryNumber>0</RegistryNumber>
|
|
<NameOfSubstance UI="D006454">Hemoglobins</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>0</RegistryNumber>
|
|
<NameOfSubstance UI="D012717">Sesquiterpenes</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>42HK56048U</RegistryNumber>
|
|
<NameOfSubstance UI="D014443">Tyrosine</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>5V2JDO056X</RegistryNumber>
|
|
<NameOfSubstance UI="D014238">Trichloroacetic Acid</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>EC 3.4.23.1</RegistryNumber>
|
|
<NameOfSubstance UI="D010434">Pepsin A</NameOfSubstance>
|
|
</Chemical>
|
|
</ChemicalList>
|
|
<CitationSubset>IM</CitationSubset>
|
|
<MeshHeadingList>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D004305" MajorTopicYN="N">Dose-Response Relationship, Drug</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D006454" MajorTopicYN="N">Hemoglobins</DescriptorName>
|
|
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D006863" MajorTopicYN="N">Hydrogen-Ion Concentration</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D066298" MajorTopicYN="N">In Vitro Techniques</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D008722" MajorTopicYN="N">Methods</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D010434" MajorTopicYN="N">Pepsin A</DescriptorName>
|
|
<QualifierName UI="Q000037" MajorTopicYN="Y">antagonists & inhibitors</QualifierName>
|
|
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D010946" MajorTopicYN="N">Plants, Medicinal</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D012717" MajorTopicYN="N">Sesquiterpenes</DescriptorName>
|
|
<QualifierName UI="Q000494" MajorTopicYN="Y">pharmacology</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D013056" MajorTopicYN="N">Spectrophotometry, Ultraviolet</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D014238" MajorTopicYN="N">Trichloroacetic Acid</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D014443" MajorTopicYN="N">Tyrosine</DescriptorName>
|
|
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
|
|
</MeshHeading>
|
|
</MeshHeadingList>
|
|
</MedlineCitation>
|
|
<PubmedData>
|
|
<History>
|
|
<PubMedPubDate PubStatus="pubmed">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
</PubMedPubDate>
|
|
<PubMedPubDate PubStatus="medline">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
<Hour>0</Hour>
|
|
<Minute>1</Minute>
|
|
</PubMedPubDate>
|
|
<PubMedPubDate PubStatus="entrez">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
<Hour>0</Hour>
|
|
<Minute>0</Minute>
|
|
</PubMedPubDate>
|
|
</History>
|
|
<PublicationStatus>ppublish</PublicationStatus>
|
|
<ArticleIdList>
|
|
<ArticleId IdType="pubmed">21</ArticleId>
|
|
</ArticleIdList>
|
|
</PubmedData>
|
|
</PubmedArticle>
|
|
<PubmedArticle>
|
|
<MedlineCitation Status="MEDLINE" Owner="NLM">
|
|
<PMID Version="1">22</PMID>
|
|
<DateCompleted>
|
|
<Year>1976</Year>
|
|
<Month>01</Month>
|
|
<Day>26</Day>
|
|
</DateCompleted>
|
|
<DateRevised>
|
|
<Year>2020</Year>
|
|
<Month>02</Month>
|
|
<Day>25</Day>
|
|
</DateRevised>
|
|
<Article PubModel="Print">
|
|
<Journal>
|
|
<ISSN IssnType="Print">0004-4172</ISSN>
|
|
<JournalIssue CitedMedium="Print">
|
|
<Volume>25</Volume>
|
|
<Issue>9</Issue>
|
|
<PubDate>
|
|
<Year>1975</Year>
|
|
<Month>Sep</Month>
|
|
</PubDate>
|
|
</JournalIssue>
|
|
<Title>Arzneimittel-Forschung</Title>
|
|
<ISOAbbreviation>Arzneimittelforschung</ISOAbbreviation>
|
|
</Journal>
|
|
<ArticleTitle>[Demonstration of tumor inhibiting properties of a strongly immunostimulating
|
|
low-molecular weight substance. Comparative studies with ifosfamide on the immuno-labile DS
|
|
carcinosarcoma. Stimulation of the autoimmune activity for approx. 20 days by BA 1, a
|
|
N-(2-cyanoethylene)-urea. Novel prophylactic possibilities].
|
|
</ArticleTitle>
|
|
<Pagination>
|
|
<MedlinePgn>1369-79</MedlinePgn>
|
|
</Pagination>
|
|
<Abstract>
|
|
<AbstractText>A report is given on the recent discovery of outstanding immunological properties in
|
|
BA 1 [N-(2-cyanoethylene)-urea] having a (low) molecular mass M = 111.104. Experiments in 214 DS
|
|
carcinosarcoma bearing Wistar rats have shown that BA 1, at a dosage of only about 12 percent
|
|
LD50 (150 mg kg) and negligible lethality (1.7 percent), results in a recovery rate of 40
|
|
percent without hyperglycemia and, in one test, of 80 percent with hyperglycemia. Under
|
|
otherwise unchanged conditions the reference substance ifosfamide (IF) -- a further development
|
|
of cyclophosphamide -- applied without hyperglycemia in its most efficient dosage of 47 percent
|
|
LD50 (150 mg kg) brought about a recovery rate of 25 percent at a lethality of 18 percent.
|
|
(Contrary to BA 1, 250-min hyperglycemia caused no further improvement of the recovery rate.)
|
|
However this comparison is characterized by the fact that both substances exhibit two quite
|
|
different (complementary) mechanisms of action. Leucocyte counts made after application of the
|
|
said cancerostatics and dosages have shown a pronounced stimulation with BA 1 and with
|
|
ifosfamide, the known suppression in the post-therapeutic interval usually found with standard
|
|
cancerostatics. In combination with the cited plaque test for BA 1, blood pictures then allow
|
|
conclusions on the immunity status. Since IF can be taken as one of the most efficient
|
|
cancerostatics--there is no other chemotherapeutic known up to now that has a more significant
|
|
effect on the DS carcinosarcoma in rats -- these findings are of special importance. Finally,
|
|
the total amount of leucocytes and lymphocytes as well as their time behaviour was determined
|
|
from the blood picture of tumour-free rats after i.v. application of BA 1. The thus obtained
|
|
numerical values clearly show that further research work on the prophylactic use of this
|
|
substance seems to be necessary and very promising.
|
|
</AbstractText>
|
|
</Abstract>
|
|
<AuthorList CompleteYN="Y">
|
|
<Author ValidYN="Y">
|
|
<LastName>Ardenne</LastName>
|
|
<ForeName>M</ForeName>
|
|
<Initials>M</Initials>
|
|
</Author>
|
|
<Author ValidYN="Y">
|
|
<LastName>Reitnauer</LastName>
|
|
<ForeName>P G</ForeName>
|
|
<Initials>PG</Initials>
|
|
</Author>
|
|
</AuthorList>
|
|
<Language>ger</Language>
|
|
<PublicationTypeList>
|
|
<PublicationType UI="D003160">Comparative Study</PublicationType>
|
|
<PublicationType UI="D004740">English Abstract</PublicationType>
|
|
<PublicationType UI="D016428">Journal Article</PublicationType>
|
|
</PublicationTypeList>
|
|
<VernacularTitle>Nachweis krebshemmender Eigenschaften einer stark immunstimulierenden Verbindung
|
|
kleiner Molekülmasse. Versuche am immunlabilen DS-Karzinosarkom im Vergleich mit Ifosfamid.
|
|
Stimulierung der körpereigenen Abwehr über etwa 20 Tage durch BA 1, einen
|
|
N-(2-Cyanthylen)-harnstoff. Neue prophylaktische Möglichkeiten
|
|
</VernacularTitle>
|
|
</Article>
|
|
<MedlineJournalInfo>
|
|
<Country>Germany</Country>
|
|
<MedlineTA>Arzneimittelforschung</MedlineTA>
|
|
<NlmUniqueID>0372660</NlmUniqueID>
|
|
<ISSNLinking>0004-4172</ISSNLinking>
|
|
</MedlineJournalInfo>
|
|
<ChemicalList>
|
|
<Chemical>
|
|
<RegistryNumber>0</RegistryNumber>
|
|
<NameOfSubstance UI="D000970">Antineoplastic Agents</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>8N3DW7272P</RegistryNumber>
|
|
<NameOfSubstance UI="D003520">Cyclophosphamide</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>8W8T17847W</RegistryNumber>
|
|
<NameOfSubstance UI="D014508">Urea</NameOfSubstance>
|
|
</Chemical>
|
|
<Chemical>
|
|
<RegistryNumber>UM20QQM95Y</RegistryNumber>
|
|
<NameOfSubstance UI="D007069">Ifosfamide</NameOfSubstance>
|
|
</Chemical>
|
|
</ChemicalList>
|
|
<CitationSubset>IM</CitationSubset>
|
|
<MeshHeadingList>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D000970" MajorTopicYN="N">Antineoplastic Agents</DescriptorName>
|
|
<QualifierName UI="Q000494" MajorTopicYN="N">pharmacology</QualifierName>
|
|
<QualifierName UI="Q000627" MajorTopicYN="Y">therapeutic use</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D002296" MajorTopicYN="N">Carcinosarcoma</DescriptorName>
|
|
<QualifierName UI="Q000188" MajorTopicYN="Y">drug therapy</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D003520" MajorTopicYN="N">Cyclophosphamide</DescriptorName>
|
|
<QualifierName UI="Q000031" MajorTopicYN="Y">analogs & derivatives</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D004353" MajorTopicYN="N">Drug Evaluation, Preclinical</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D004361" MajorTopicYN="N">Drug Tolerance</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D004906" MajorTopicYN="N">Erythrocyte Count</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D006863" MajorTopicYN="N">Hydrogen-Ion Concentration</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D006943" MajorTopicYN="N">Hyperglycemia</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D007069" MajorTopicYN="Y">Ifosfamide</DescriptorName>
|
|
<QualifierName UI="Q000494" MajorTopicYN="N">pharmacology</QualifierName>
|
|
<QualifierName UI="Q000627" MajorTopicYN="N">therapeutic use</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D007109" MajorTopicYN="N">Immunity</DescriptorName>
|
|
<QualifierName UI="Q000187" MajorTopicYN="Y">drug effects</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D007165" MajorTopicYN="N">Immunosuppression</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D007928" MajorTopicYN="N">Lethal Dose 50</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D007958" MajorTopicYN="N">Leukocyte Count</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D051379" MajorTopicYN="N">Mice</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D009374" MajorTopicYN="N">Neoplasms, Experimental</DescriptorName>
|
|
<QualifierName UI="Q000188" MajorTopicYN="N">drug therapy</QualifierName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D051381" MajorTopicYN="N">Rats</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D013268" MajorTopicYN="N">Stimulation, Chemical</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D013997" MajorTopicYN="N">Time Factors</DescriptorName>
|
|
</MeshHeading>
|
|
<MeshHeading>
|
|
<DescriptorName UI="D014508" MajorTopicYN="N">Urea</DescriptorName>
|
|
<QualifierName UI="Q000031" MajorTopicYN="Y">analogs & derivatives</QualifierName>
|
|
<QualifierName UI="Q000494" MajorTopicYN="N">pharmacology</QualifierName>
|
|
<QualifierName UI="Q000627" MajorTopicYN="N">therapeutic use</QualifierName>
|
|
</MeshHeading>
|
|
</MeshHeadingList>
|
|
</MedlineCitation>
|
|
<PubmedData>
|
|
<History>
|
|
<PubMedPubDate PubStatus="pubmed">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
</PubMedPubDate>
|
|
<PubMedPubDate PubStatus="medline">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
<Hour>0</Hour>
|
|
<Minute>1</Minute>
|
|
</PubMedPubDate>
|
|
<PubMedPubDate PubStatus="entrez">
|
|
<Year>1975</Year>
|
|
<Month>9</Month>
|
|
<Day>1</Day>
|
|
<Hour>0</Hour>
|
|
<Minute>0</Minute>
|
|
</PubMedPubDate>
|
|
</History>
|
|
<PublicationStatus>ppublish</PublicationStatus>
|
|
<ArticleIdList>
|
|
<ArticleId IdType="pubmed">22</ArticleId>
|
|
</ArticleIdList>
|
|
<ReferenceList>
|
|
<Reference>
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|
<Citation>Crit Care Med. 2006 Jul;34(7):1913-7</Citation>
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|
<ArticleIdList>
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<ArticleId IdType="pubmed">16715038</ArticleId>
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</ArticleIdList>
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</Reference>
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<Reference>
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<Citation>Crit Care. 2007;11(2):R31</Citation>
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|
<ArticleIdList>
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|
<ArticleId IdType="pubmed">17331245</ArticleId>
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</Reference>
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<Reference>
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<Citation>J Am Soc Nephrol. 2010 Feb;21(2):345-52</Citation>
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<ArticleIdList>
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</Reference>
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<Reference>
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<Citation>Am J Kidney Dis. 2010 Oct;56(4):651-60</Citation>
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<ArticleIdList>
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<ArticleId IdType="pubmed">20673605</ArticleId>
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</Reference>
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<Reference>
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<Citation>Blood Purif. 2010;30(2):120-6</Citation>
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<ArticleIdList>
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</Reference>
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<Reference>
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<Citation>Nephrol Dial Transplant. 2011 Jul;26(7):2161-8</Citation>
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</Reference>
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<Reference>
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<Citation>Am J Kidney Dis. 2011 Feb;57(2):228-34</Citation>
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</Reference>
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<Reference>
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<Citation>Clin J Am Soc Nephrol. 2012 Apr;7(4):533-40</Citation>
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<ArticleIdList>
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<ArticleId IdType="pubmed">22362062</ArticleId>
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</Reference>
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<Reference>
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<Citation>QJM. 2012 Aug;105(8):729-40</Citation>
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<ArticleIdList>
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</ArticleIdList>
|
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</Reference>
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<Reference>
|
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<Citation>Clin J Am Soc Nephrol. 2012 May;7(5):712-9</Citation>
|
|
<ArticleIdList>
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<ArticleId IdType="pubmed">22422536</ArticleId>
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</Reference>
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<Reference>
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<Citation>QJM. 2013 Apr;106(4):323-32</Citation>
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<ArticleIdList>
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<ArticleId IdType="pubmed">23345468</ArticleId>
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</ArticleIdList>
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</Reference>
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<Reference>
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<Citation>J R Coll Physicians Edinb. 2013;43(1):37-8</Citation>
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<ArticleIdList>
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<ArticleId IdType="pubmed">23516690</ArticleId>
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</ArticleIdList>
|
|
</Reference>
|
|
</ReferenceList>
|
|
</PubmedData>
|
|
</PubmedArticle>
|
|
<PubmedArticle>
|
|
<MedlineCitation Status="MEDLINE" Owner="NLM">
|
|
<PMID Version="1">23</PMID>
|
|
<DateCompleted>
|
|
<Year>1976</Year>
|
|
<Month>01</Month>
|
|
<Day>26</Day>
|
|
</DateCompleted>
|
|
<DateRevised>
|
|
<Year>2020</Year>
|
|
<Month>02</Month>
|
|
<Day>25</Day>
|
|
</DateRevised>
|
|
<Article PubModel="Print">
|
|
<Journal>
|
|
<ISSN IssnType="Print">0004-4172</ISSN>
|
|
<JournalIssue CitedMedium="Print">
|
|
<Volume>25</Volume>
|
|
<Issue>9</Issue>
|
|
<PubDate>
|
|
<Year>1975</Year>
|
|
<Month>Sep</Month>
|
|
</PubDate>
|
|
</JournalIssue>
|
|
<Title>Arzneimittel-Forschung</Title>
|
|
<ISOAbbreviation>Arzneimittelforschung</ISOAbbreviation>
|
|
</Journal>
|
|
<ArticleTitle>Effect of etafenone on total and regional myocardial blood flow.</ArticleTitle>
|
|
<Pagination>
|
|
<MedlinePgn>1400-3</MedlinePgn>
|
|
</Pagination>
|
|
<Abstract>
|
|
<AbstractText>The distribution of blood flow to the subendocardial, medium and subepicardial layers
|
|
of the left ventricular free wall was studied in anaesthetized dogs under normoxic (A), hypoxic
|
|
(B) conditions and under pharmacologically induced (etafenone) coronary vasodilation (C).
|
|
Regional myocardial blood flow was determined by means of the particle distribution method. In
|
|
normoxia a transmural gradient of flow was observed, with the subendocardial layers receiving a
|
|
significantly higher flow rate compared with the subepicardial layers. In hypoxia induced
|
|
vasodilation this transmural gradient of flow was persistent. In contrast a marked
|
|
redistribution of regional flow was observed under pharmacologically induced vasodilation. The
|
|
transmural gradient decreased. In contrast to some findings these experiments demonstrate that a
|
|
considerable vasodilatory capacity exists in all layers of the myocardium and can be utilized by
|
|
drugs. The differences observed for the intramural distribution pattern of flow under hypoxia
|
|
and drug induced vasodilation support the hypothesis that this pattern reflects corresponding
|
|
gradients of regional myocardial metabolism.
|
|
</AbstractText>
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<MeshHeading>
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<ArticleId IdType="pubmed">19140997</ArticleId>
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<Citation>Clin Med (Lond). 2009 Oct;9(5):417-20</Citation>
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<ArticleId IdType="pubmed">19886098</ArticleId>
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|
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<ISSN IssnType="Print">0003-6919</ISSN>
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<Volume>19</Volume>
|
|
<Issue>6</Issue>
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<PubDate>
|
|
<Year>1970</Year>
|
|
<Month>Jun</Month>
|
|
</PubDate>
|
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</JournalIssue>
|
|
<Title>Applied microbiology</Title>
|
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|
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|
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<ArticleTitle>Bactericidal activity of a broad-spectrum illumination source.</ArticleTitle>
|
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<Pagination>
|
|
<MedlinePgn>1013-4</MedlinePgn>
|
|
</Pagination>
|
|
<Abstract>
|
|
|
|
<AbstractText>Several hours of exposure to Vita-Lite lamps, which have a unique spectral
|
|
distribution, give significant killing of cells of Staphylococcus aureus.
|
|
</AbstractText>
|
|
</Abstract>
|
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<Author ValidYN="Y">
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<LastName>Himmelfarb</LastName>
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|
|
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|
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</Author>
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<Author ValidYN="Y">
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|
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